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A Bloody Serious Pressure * Her Research Made the Journal Cover * Researchers Fighting Covid-19 *
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Department of Experimental Medical Science, Newsletter 6, June 18, 2020 | |
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#atwork Photo: Agata Garpenlind
| New, nearby horizons awaiting. Ystad sandskog, less than an hours drive to the south east from Lund.
Photo: Agata Garpenlind
| Dear EMV Members,
Summer is here and it is soon time to take vacation! This year the summer break is extra welcome considering the tough times we have faced this semester. In particular, our teachers, course leaders, examiners and programme administrators have carried a large burden this spring and they cannot be thanked enough for quickly and effectively shifting to digital forms of teaching. Still, life at EMV as well as in society at large is and will continue to be characterized by Covid-19. Please keep yourselves updated through the University's website and information from the faculty. It is extremely important that we continue to follow the rules of conduct that apply, including social distancing. Accordingly, the strawberry feast had to be cancelled this year, but I do hope that we will be able to gather for “glögg” in December. The salary revision has now been completed and new salaries will be dispersed together with the June salary. The first step towards a new salary is the staff appraisal meeting and this should be held before the end of June. The aim of such meeting is to have a dialogue about the work situation and to discuss thoughts, ideas and wishes about the working environment, skills, development, tasks and the relationship with colleagues and the manager. In fact, the staff appraisal is one of the best tools to prevent conflicts and illness. If conflicts nevertheless arise that cannot be handled within the research group, please contact EMV:s work environment coordinators/HR for further help and do so early on! I want to congratulate a bunch of people: Fredrik Johansson who has been hired as associate senior lecturer; Karin Tran Lundmark and Anders Rasmussen who have been promoted to docents; Lena Uller and Tomas Deierborg of have been/soon will be promoted to professors and Åsa Petersén who has been selected as Wallenberg Clinical Scholar. Well done all of you! All in all, EMV continues to grow. We are now around 380 people at EMV, disseminated over 70 independent research groups. Recruitment is ongoing for three teacher positions (lectureship in morphology, lectureship in molecular imaging and professorship in immunology). While I am very glad to greet new people to EMV I am equally sad to have to say goodbye to some. Tomas Leanderson is about to retire, and he has had a fabulous career. Please read more further down! Jens Lagerstedt is leaving EMV for new and great opportunities at Novo Nordisk. I wish you both all the best! I will also take on new challenges. From August 15, I will be the The Swedish Research Council’s (Vetenskapsrådet) new Secretary General for Medicine and Health. However, I will remain as Head of Department (50%) until December 31. The EMV nomination committee has started their work selecting a new head of department. They will soon reach out to you (but feel free to contact Angela Cenci-Nilsson and/or Lisette Eklund at emv-valberedning@med.lu.se). I kindly ask you to bear with me during this fall as I might not answer e-mails as quickly as I usually do! Typically, I will be in Stockholm Monday-Tuesday and in Lund Wednesday-Friday. I now wish you all a great and relaxing holiday. Summer is by no means cancelled. Instead embrace staycation! Madde | Madeleine Durbeej-Hjalt, Professor and Head of Department
at the Department of Experimental Medical Science. Photo: Agata Garpenlind
| | | | Oxana Klementieva's research made the front cover of the March issue of Advanced Science. This cover illustrates a novel approach for molecular imaging of amyloid aggregates in the neuronal cell by laser-focused infrared light. Thanks to the high resolution of the optical photothermal infrared t, it is now possible to image molecular structures directly in the neuron at subcellular resolution.
| New Imaging Method Sheds Light on Alzheimer's DiseaseTo understand what happens in the brain when Alzheimer's disease
develops, researchers need to be able to study the molecular structures
in the neurons affected by Alzheimer's disease. Researchers at Lund
University in Sweden have tested a new imaging method for this purpose.
The research is published in the journal Advanced Science.
By Tove Smeds
In Alzheimer's disease (AD),
so-called beta-amyloid plaques are formed in the brain, and neurons in
the brain die. When the plaque becomes visible in brain tissue, the
disease is already in an advanced stage. As more and more nerve cells
die, memory disruptions begin, something that eventually results in
memory loss. But what happens in the nerve cells before amyloid plaques
appear, and why do nerve cells die?
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Oxana Klementieva and her research group, from the left: Stefan Broselid, Postdoctoral researcher, Agnieszka Agnes Paulus and Nadja Gustavsson, PhD students, Oxana Klementieva, PI Medical Microspectroscopy,, Flora Haddy, Masters student. Photos: Agata Garpenlind | – This is a question that
researchers have long struggled to find answers to. We have not had
enough imaging techniques to study the structural changes in nerve
cells. This is required to detect very early changes, and therefore
potentially understand the triggers”, says researcher Oxana Klementieva,
group leader for medical microscopy at Lund University. Using a new method, optical photothermal spectroscopy, O-PTIR... continue to full article
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Using novel label-free optical photothermal infrared super-resolution imaging, amyloid protein aggregates can be detected directly in AD affected neurons at the subcellular level. |
Image of a human embryo surrounded by placenta, around 7 weeks of age.
(Hill, M.A. (2020, May 18) Embryology Stage13 bf4.jpg. (Retrieved from here) |
New Method Provides Unique Insight Into the Development of the Human BrainStem
cell researchers at Lund University in Sweden have developed a new
research model of the early embryonic brain. The aim of the model is to
study the very earliest stages of brain to understand how different
regions in the brain are formed during embryonic development. With this
new insight, researchers hope to be able to produce different types of
neural cells for the treatment of neurological diseases more
efficiently. The study has been published in the journal Nature Biotechnology.
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Agnete Kirkeby, researcher at the
Wallenberg Centre for Molecular Medicine and the Department of
Experimental Medical Science. Photo: Kennet Ruona
| | By Agata Garpenlind In order to
develop stem cell treatments for neurological diseases such as
Parkinson’s Disease, epilepsy and stroke, researchers must first
understand how the human brain develops in the embryonic stage. With
knowledge of how neural cells are formed at different developmental
stages, researchers have the opportunity to develop new stem cell
therapies more quickly in the laboratory. – The challenge is that
there are thousands of different sub-types of neural cells in the human
brain, and for each disease we need to be able to produce exactly the
right type of neural cell”, says Agnete Kirkeby, researcher at the
Wallenberg Centre for Molecular Medicine and the Department of
Experimental Medical Science at Lund University. Studies
on how each individual neural cell forms in the embryo during brain
development are essential for the researchers to be able to understand
how to produce these specific cells in the laboratory. Research
on the early development of the human brain, from five days after the
fertilisation of the cell to approximately seven weeks, have so far been
difficult... continue to full article | | | | Professor of Immunology, Tomas Leanderson, here with pals Hasse and Sölve, will retire July 1. Photo: Agata Garpenlind |
One of the Last Ones Out of the Old School Professors
Doesn´t Want to Shuffle Around
the Hallways Like a Ghost
A
sun drenched day in the allies of Västra Hamnen. A whirlwind of black
and white tails, snouts and barks are on duty at the townhouse door. Dog
dad, Professor of Immunology Tomas Leanderson, hushes, admonishes,
welcomes into the kitchen table and coffee. In a few days, this rare
specimen of the old kind of professor at the Faculty of Medicine will be
a free man, after a life-long love affair with immunology.
The opinions shared in this article are Tomas Leandersons´.
By Agata Garpenlind
Two moments in a day are special. One is early, early, at five, six, a
weekend morning. A cup of coffee, black, the print newspaper spread out
over the kitchen table. The flock of the house; people, dogs, asleep.
Only Leanderson´s awake. — And you feel that everyone is safe, everyone is calm, and that this moment is just mine. The second moment: a weekday morning. —
I´ve been out walking the dogs, my wife has gone to work, the kids are
in school. And I can have another sup of coffee, and just sit there, not
looking at the clock, and think, "So, what should I be thinking about
today?". On July 1, Tomas Leanderson will retire from his job as a
Professor of Immunology at the Faculty of Medicine, after 30 years on
the chair and over 40 years as a researcher. — My professional life has
been extremely fun and fulfilling, but it´s only when you finish
something completely that you can really start something new. If you
stay part-time, there will be no new beginning.He has been planning
for his exit for a long time now.
— I´ve been responsible for a great
many people for most of my professional life. It´ll be nice to take a
break from those responsibilities. I´ve always been against that the old
professors wander around like old ghosts in the university corridors.
They become a stopping block for change, even if they don’t believe it
themselves. He believes in idleness, boredom, as a source of creativity. — So now I just have to wait until I get really bored... ... continue to full article in English ... continue to original full article in Swedish | Leandersons tips for new researchers:- One
should choose to become a researcher based on desire. You really should
feel that you are passionate about research because it is a very lonely
job with few gratifications in many ways.
- Dare to go your own
way. And to do that, you have to act independently of others'
affirmations and pats on the shoulder. Confidence needs to come from
within yourself.
- Move around and see different environments
within the frame of your work. It is good for research to see how
different people think. It´s good for your creativity.
| | | Retires on July 1st at age 64 Family: Wife Karin, four
children. Two live at home now, two are adults. 3 dogs: Hasse, Gösta
and Sölve, Danish-Swedish farm dogs Lives: In a townhouse in the shadow of Turning Torso in Västra Hamnen in Malmö
| | | Accumulating levels of protein aggregates characteristic of Huntington's disease (left) can be reduced by boosting autophagy (right). Image
source: Karolina Pircs.
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Autophagy and the Treatment of Huntington´s Disease: Timing is Key
Huntington
disease is fatal inherited brain disorder, usually emerging during
mid-life. Although there is currently no known cure, the Molecular
Neurogenetics group at Lund Stem Cell Center aims to understand the
defective cellular processes leading to the progression of this disease.
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Researcher Karolina Pircs. Photo: Private
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– This model exhibits a severe and rapidly developing form of mutant HTT protein accumulation. We
can tightly control many aspects of this model, making it a powerful
tool for understanding the mechanisms involved in Huntington disease
progression. .... continue to full article | | By Alexander Doyle Characterised by symptoms including impaired motor and
cognitive function, Huntington disease is caused by the expansion of a
CAG trinucleotide repeat in the huntingtin (HTT) gene. This results in
the production of an abnormal mutant HTT protein, which forms aggregates
in the brain, leading to neurodegeneration and driving disease
progression. Autophagy, a mechanism
employed by the cell to remove unnecessary or disfunctional components,
has been reported as essential for the degradation of the mutant HTT
protein aggregates.
However, the degree of involvement of this process
in disease progression is a controversial topic. In her first paper as
senior author, Dr Karolina Pircs hopes to shed some light on this long-standing debate. – In
this paper we used a mouse model of Huntington disease, where we inject
neuron specific viral vectors into the striatum to drive the
over-expression of either wild type, or mutant HTT, explains Karolina.
| | | | Researcher Darcy Wagner. Photo: Agata Garpenlind
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Two Researchers at EMV fight Covid-19 With Support From SciLifeLabDuring this past pandemic spring, SciLifeLab, together with the Knut and Alice Wallenberg Foundation, launched a comprehensive national program with 67 research projects dealing with covid-19. The purpose is to fight the corona pandemic and in total the projects share 50 million SEK. Six research projects have been part of the grant at Lund University. Two of them are researchers at EMV: Darcy Wagner and Sonja Aits.
By Tove Smeds
SARS-CoV-2 infects the cells of the
respiratory tract via the so-called ACE receptors. Human lungs are very
different from other species commonly used in the biomedical research, which
makes it more difficult when researchers want to set up models to study how an
infected lung responds to different drugs. – This becomes particularly clear when it comes to zoonotic viruses, ie a virus
transmitted from animals to humans, such as SARS-CoV-2. The virus can be
completely passive in an animal species, but infect human tissue. We need to
understand more about what happens in human lung tissue and how the virus
causes disease, and for that human models are needed, says Darcy Wagner, WCMM researcher
at Lung bioengineering and regeneration at Lund University.
With the support of SciLifeLab, Darcy Wagner and
colleagues will develop a model in which they allow the virus to infect human
lung... continue
to full article about Darcy Wagners study
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Researcher Sonja Aits. Photo: Private
| | In the first
week of May this year alone, 4,000 scientific articles on the disease
were published - an amount that makes it difficult for healthcare
professionals, policy makers and researchers to stay up to date. Sonja Aits and her research group are now developing text analysis tools
that use artificial intelligence to find relevant information. The AI system can also extract key information about, for example, links between a drug and the disease, or between the virus and various human proteins, which can be valuable when researchers search for possible treatments. No humaan being can keep bits and pieces from thousands of articles in his or her head, says Sonja Aits...continue to full article (in Swedish) about Sonja Aits´study.
| | | | Photos from the lab of Cellular Biomechanics,
research group at the Department of Experimental Medical Science. Photos: Agata Garpenlind |
High Blood Pressure is Bloody Serious
High
blood pressure damages our blood vessels. It also damages our heart,
our brain and our kidneys. A high blood pressure increases the risk of
suffering a heart attack and stroke. However, it is not known exactly
why a high blood pressure is so dangerous. – The reason is that we have effective antihypertensive medications. So
why bother how pressure-induced damage occurs. We feel that this
position needs to be re-evaluated, says Karl Swärd, professor
of physiology, who leads the Cellular Biomechanics research group at
Lund University. The group wants to find answers to how an elevated
blood pressure damages the vessels.
By Agata Garpenlind
Hypertension, that is, an elevated blood pressure, recently overtook
first place among risk factors for the world's overall disease burden. –
The more other risk factors an individual has for heart and vascular
disease, the more reason to control and treat their elevated blood
pressure, says Karl Swärd. His
research group, Cellular Biomechanics, at the Department of Experimental
Medical Science in Lund wants to find out how pressure is sensed and
decoded in cells of the vascular wall. This is done in order to
understand the mechanisms underlying the harmful effects of blood
pressure on the vessels.– If we can identify the underlying causes,
we can hopefully find new mechanisms that can be targeted to prevent or
repair mechanically induced vascular damage. .... continue to full article |
Cellular Biomechanics, from the left: Li Liu, Katazyna Kawka, Catarina Rippe, Karl
Swärd and Francesco de Luca. Photo: Agata Garpenlind
| Karl Swärd is the research
group leader for Cellular Biomechanics. Karl Swärd studied at the medical program in Lund
between 1989 and 1992 and then began his research career. He received
his PhD in 1997 and then did a postdoc in Canada. In 2007 he received
Senior Scientist position at the Swedish Research Council and now he is a
professor at the Faculty of Medicine in Lund. Photo: Agata Garpenlind | | | |
The
research field cellular biomechanics deals with how chemical energy is
converted into mechanical work and the opposite, that is, how mechanical
signals are perceived and converted into a biological response. These
processes are fundamental and critical to life. These processes also
play a central role in the development of several different medical
conditions; atherosclerosis, heart disease and cancer. One tissue that
the group studies intensively is the vascular wall that is subjected to heavy
mechanical load (blood pressure, and so-called shear forces
(deformations without volume change). This mechanical load is likely a
major contributor to the development of atherosclerosis. The group consists of: Karl Swärd, 51, PhD in Physiology, leads the research group Catarina Rippe, 48, PhD in Medical Chemistry Li Liu, 30, doctor and doctoral student Francesco de Luca, 43, biomedical student and phD student Katarzyna Kawka, 51, BMA, (shared with Sebastian Albinsson and Bengt-Olof Nilsson)
| Isolated lymphoid follicle of the human large intestine. Cells were stained in different colours, T
cells (white), B cells (red), and germinal centers (green). T and B
cells are two key immune cell types, germinal centers are important for
the generation of antibody responses by B cells. |
New Knowledge About Our Immune System Provided by Novel Technique |
Professor William Agace. Photo: Agata Garpenlind
| Using
these techniques, they described
the distribution of GALT along the length of the human intestine and
performed a detailed phenotypic and functional analysis of the adaptive
immune cell compartment of human GALT as well GALT-free lamina propria.
– Our findings provide a first ‘roadmap’
of the GALT structure and composition along the length of the human
intestine and additionally suggest that human GALT play an important
role in driving region-specific adaptive immune responses within the
intestine”, Professor William Agace says.
Continue to full article
This article was first published at dtu.dk | | New
unique techniques for investigating our intestinal immune system can
lead to new possibilities for treatment of chronic intestinal diseases.
The
intestinal immune system is essential for gut health, but when
dysregulated it can lead to chronic diseases such as Crohn’s disease and
ulcerative colitis.
The intestinal immune system can be divided into two functionally distinct immune compartments:
- the Gut-Associated Lymphoid
Tissues (GALT), where adaptive immune cells are initially activated and
differentiate into specific functional subsets;
- and the intestinal lamina propria, where activated immune cells localize and perform their effector and regulatory functions.
However, our current understanding of
the immune composition and function of human GALT is limited, due to a
lack of protocols to identify and isolate these compartments free from
contaminating tissues.
A research team headed by Professor William Agace, DTU Health Tech, have
developed unique techniques to identify and isolate human GALT.
| | | | Illustration: Lisa Karlsson
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Identification of Prognostic Markers for Development of Chronic Lung Fibrosis in COVID-19 Patients
Pulmonary
fibrosis is a severe and life shortening chronic condition. Although
most people experience mild symptoms of COVID-19, some patients develop
serious and life-threatening conditions with severe lung damage. In
order to influence the outcome of the disease, lung researchers in Lund
are currently establishing methods to identify risk groups at an early
stage. | | | |
Gunilla
Westergren-Thorsson, Professor of Lung Biology Photo: Agata Garpenlind
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These potential biomarkers will be further studied in blood
samples from patients with pulmonary fibrosis and patients who have had a
COVID-19 infection, in order to characterize COVID-19 patients at risk
of developing pulmonary fibrosis. – We will link our results to clinical parameters such as ... continue to full article
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By Agata Garpenlind A
significantly altered lung structure with an overproduction of
connective tissue, so-called pulmonary fibrosis, is a chronic condition
with profound suffering and impaired quality of life. In its most severe form, the lungs stop working with a premature death as a result. In severe COVID-19 infection the lung tissue composition become seriously affected with massive inflammation and fibrosis. – We need methods for early diagnosis of patients likely to develop fibrotic changes. Being able to identify risk groups will help us to start treating these patients at an early stage" says Gunilla
Westergren-Thorsson, Professor of Lung Biology at the Department of
Experimental Medical Science at Lund University, who is leading the
study. When studying
fibrotic lung tissue in the lab, researchers have found proteins, unique
markers, reflecting the clinical course seen in patients with pulmonary
fibrosis.
| | | |
Facts about the study
Name: "Identifying early signs of permanent lung damage in COVID-19 patients” Started 2020-05-07 The Lung biology team consists
of about 20 people led by Professor Gunilla Westergren-Thorsson. The
group aims to contribute to a better understanding of the mechanisms
behind chronic inflammatory lung diseases. The aim is also to identify
biomarkers for early diagnosis and new targets for treatment of these
diseases. | Huntington researcher Åsa Petersén. Photo: Agata Garpenlind |
Huntington's Disease – a Fascinating And Touching MysteryA
person who carries the mutant gene will at some point in his or her
life develop the deadly Huntington's disease. This brain disease can be
inherited from generation to generation and begins insidiously, making
it increasingly difficult to regulate emotions, thoughts, then
movements. There is no treatment that slows down the disease. But
Huntington researcher and psychiatrist Åsa Petersén works hard at it.
– As a doctor, I feel despair when I see my patients deteriorate
without being able to slow down the process. But it also gives me strong
motivation to continue to work on it, she says.
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Åsa Petersén working at the clinic. Photo: Agata Garpenlind
| to really improve the quality of life and
survival of these families.So she's doing research. Her group's research is known as translational. This means... continue to full article
| | By Agata Garpenlind We meet at one of her workplaces, the Huntington Disease Clinic, at
the Neurology Clinic at Skåne University Hospital, SUS, in Lund, high up
in Blocket. Hazy views of rapeseed fields, all the way to the sea, blue
and beautiful. This is where Åsa Petersén meets the affected
individuals and their families. – In my work I´m driven by the need I
see when I meet the affected families. It´s clear that the psychiatric
symptoms are the most difficult to deal with for the affected
individuals and their loved ones. It´s very sad to see how the disease
impairs a person's function over time. It becomes crystal clear that
more research is needed
| | | | Åsa Petersén, Professor of Neuroscience and
consultant in psychiatry, heads the Huntington Center at Lund
University. She has recently been appointed Wallenberg Clinical Scholar
in 2020 with a research grant of 15 million SEK over five years to find
methods that can slow the progression of Huntington's disease. |
Seven Principles for
Good Public Research Communication
As
a researcher or communications officer, when you are to convey
information about research via the media, social media, events or in
other public contexts, you should strive to observe the seven principles
below.
As
a university, our mission includes external engagement and providing
information about our activities and working for our research findings
to be utilised. It is in the interests of the researchers, the
University and society that findings and insights on the methodology
and limitations of research are made accessible for public debate,
political decisions, the business community and society as a whole.
- Correctness. When research is communicated, the content must be correct and give the
target group a correct understanding of the relevant facts about both
the results and the research involved.
- Relevance. What
is relevant for the recipient to know? Bear in mind that the information
conveyed is to be relevant for the recipient’s understanding of the
research results. Provide relevant information about facts and data to
ensure that content and conclusions cannot be misinterpreted or
overinterpreted. Avoid communicating alarming messages unnecessarily.
- Uncertainty. Is
there any uncertainty? Clarify the uncertainties that relate to the
research. Scientific discoveries are associated with varying uncertainty
factors depending on method, research design, data or theoretical
suppositions. Be clear about any limitations/weaknesses/uncertainties
relating to the results and their interpretation.
... continue reading
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EMV's administrations manager, Sofie Magnusson, gets
connected. Photo: Agata Garpenlind
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Digital Workplace Meetings, Coffee at a Distance
and Handy Desinfectants
Every Thursday, at ten o'clock, the Administration
at EMV gets together for a workplace meeting (APT). Now, in the age of Corona, the
meeting is still at ten, but partly at a distance.
By Agata Garpenlind
It´s almost ten o´clock. All employees who are on
site at BMC on this particular Thursday morning are spreading out in chairs and
sofas in the open-space canteen at I13. continue reading
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UniStem Day 2020. Photos: Alexander Doyle
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Bringing Stem Cell Research to the Next Generation of ScientistsUniStem
Day 2020 was another great success, with high school students visiting
Lund University to enjoy a variety of activities introducing them to the
fascinating world of stem cell research.
By Alexander Doyle
The 6th UniStem day hosted by Lund University
kicked off at the LUX Aula lecture theatre, Lund University, where over
200 high school students from over the Skåne region were welcomed by
event co-ordinator Assistant Prof. Göran Karlsson and Stem Cell Center Director Prof. Zaal Kokaia. During the first session of the day, Dr Mattias Magnusson, principal investigator from the Stem Cell and Cancer Stem Cell Regulation group, introduced the... continue reading
| New Publications, Dissertations & Halftime Reviews
The publications listed below are the most recent by researchers at the department at this newsletters date of publication. Follow the links below. | Staffing at communications during summer Who is in place and when at the Medfaks central communications department is specified here. During four summer weeks no-one or very few will be on duty. In this case the following applies: - In case of emergency, call Head of Communications Johanna Sandahl on 0709943479.
- For cases that are not urgent, but cannot wait until after the holidays: email info@med.lu.se This adress gets checked every weekday throughout the summer.
EMV´s Communications Officer is out of office June 22 and July 17 and August 10 through 14. |
|
Invoices in the system Proceedo/Lupin that are
not correct
If
something is wrong with the invoice you have received in the system
Proceedo/Lupin, tick the box “Investigation ongoing” and then Save for
later.
| | | | Then call the supplier and ask for a credit invoice. Once you have
received the credit invoice you can approve both invoices. NB! Include a
comment. These options are available in the
top right-hand corner.
This is
important since otherwise the invoice will continue to the “next level”
at our department after a couple of days for approving. | New procedure for health promotion
reimbursement in 2020
Previously, health promotion reimbursement has been paid for receipts dated a
maximum of one year back in time, provided that you apply for reimbursement for
the year in which the purchase was made. Only purchases made in November or
December can be carried over to the next year.
In 2020, new
rules have been introduced, which entail that you must have registered your case in Primula by 15
January 2021 in order to apply for reimbursement for activities purchased
and paid for in 2020. After that date,
you can no longer apply for reimbursement for activities in 2020.
Lucat during summer and vacations
If you require help with Lucat matters during July and
August, please send an e-mail to emv-lucat@med.lu.se
This address is not checked on a daily basis due to
vacation, so if you already know of persons visiting your research group during
summer, please send information now. The information required is:
First Name
Last Name
Birth date (or Swedish personal number)
Time period
Group (name or cost center)
Role (guest researcher, post doctor, PhD student, BMA
etc)
Room number and telephone number
Financing (employed by LU, external stipend, LU
stipend etc)
Safety
Inspections Fall 2020
Thursday 10/9 am: BMC D10 and D11
Wednesday 30/9 am: BMC D12 and D13
Thursday 22/10 am: BMC D14, I12 and I13
Thursday 12/11 am: BMC building F, and
Clinical Chemistry and Pharmacology
Wednesday 9/12 all day: MV
New
agreement about preparedness in case of spillage
Read more about it here
New
checklist in case of threats and violence
Read the checklist
Hazardous (chemical) waste removal during summer
During weeks 27-32, from 29 June to 9 August, there will be no regular hazardous waste removal. If you require waste removal during the break, the simplest solution is to call the Sysav switchboard +46 40 635 18 00 and select the options for "company" (företag), "hazardous waste" (farligt avfall) or email your request to order.kemi@sysav.se. More info Newsletter
on work environment, environment, security, radiation safety, fire safety and
chemical safety The recent newsletter from
LU Bygg highlights some important information regarding new rules about hazardous
goods, demanding that staff members involved in handling, packaging, and
shipping must be certified by passing a qualifying course, the importance of having staff members
at place in the laboratory when SYSAV arrives to pick up chemical waste, online course to refresh the fire
safety knowledge (N.B., this course is not a substitute for the compulsory
hands-on fire safety training). Read the whole newsletter here
COVID-19, continous updates at LU regarding the pandemic Keep
yourself continuously posted about the coronavirus outbreak through this
LU-link
|
About the EMVNewsletterThe newsletter from the Department of Experimental Medical Science is sent out to all staff and co-workers two to three times every semester. It is not possible to unsubscribe. Do you want to reach out to your co-workers with relevant information in coming issues of the newsletter?
Please e-mail your text in English to Communications Officer Agata Garpenlind, agata.garpenlind@med.lu.se, and attach the relevant web link and photos. The next newsletter is due September 10, 2020. Please provide your contribution as soon as possible, but no later than by September 3. Disclaimer: It is the EMV board, in collaboration with the communications officer, who, according to relevance, decide what material gets to be published, and when.
If you receive this newsletter but have no connection to the department, please contact your local LUCAT administrator.
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